|
Haplotype analysis of the BRAF gene in our German case-control cohort. |
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| Total |
Male |
Female |
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|
|
|
|
|
||||||
| Haplotype |
Case (N = 1000) |
Control (N = 898) |
Case (N = 470) |
Control (N = 434) |
Case (N = 530) |
Control (N = 464) |
|||
| CCTG (H1) |
84%(829) |
86%(771) |
81%(380) |
86%(374) |
85%(449) |
86%(397) |
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| ATGA (H2) |
8%(82) |
7%(59) |
p = 0.206 |
11%(52) |
7%(30) |
p = 0.040 |
6%(30) |
6%(29) |
p = 0.796 |
| CTTA (H3) |
6%(64) |
6%(58) |
5%(24) |
6%(26) |
8%(40) |
7%(32) |
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| CTTG (H4) |
2%(20) |
0%(3) |
χ2 = 16.75 p = 0.019 |
2%(11) |
0%(2) |
χ2 = 15.85 p = 0.015 |
2%(9) |
0%(1) |
χ2 = 7.82 p = 0.25 |
| CCGG (H5) |
0%(1) |
0%(4) |
0%(0) |
0%(2) |
0%(1) |
0%(2) |
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| ACTG (H6) |
0%(1) |
0%(2) |
0%(0) |
0%(0) |
0%(1) |
0%(2) |
|||
| CCTA (H7) |
0%(2) |
0%(1) |
0%(2) |
0%(0) |
0%(0) |
0%(1) |
|||
| ACGA (H8) |
0%(1) |
0%(0) |
0%(1) |
0%(0) |
0%(0) |
0%(0) |
|||
Meyer et al. Journal of Carcinogenesis 2003 2:7 doi:10.1186/1477-3163-2-7 |
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